Epstein-Barr virus rewires host epigenomes to drive stomach cancer
Researchers in Japan and Singapore have discovered a molecular mechanism that explains how Epstein-Barr virus (EBV) infection alters a host's epigenome to promote tumorigenesis (the transformation of normal cells into cancer cells) in certain types of stomach cancer.
The findings suggest that EBV infection plays an important role in the development of EBV-associated stomach cancers, and provide fresh insights that may lead to new therapies for stomach and other virus-related malignancies.
This achievement was published in Nature Genetics.
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